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After amyloid plaques form, tau and alpha-synuclein pathology rises in mice

Written by David Miller

The aging brain frequently harbors multiple protein pathologies that contribute to Alzheimer's, Parkinson's and other neurodegenerative diseases. But how do these problematic proteins interact with each other, and could these interactions inform future therapies? Researchers at TGen, part of City of Hope, are answering some of these questions with the help of a unique mouse model that combines different mixtures…

Source: Medical Xpress

About the author

David Miller

a pharmacist, a tech enthusiastic, who explored the Internet to gather all latest information pharma, biotech, healthcare and other related industries.

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